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In contrast to cyp3a-mediated metabolism and bioactivation of trazodone, formation of gsh adducts of m -cpp was found to be specifically mediated by cyp2d6, suggesting a possible relevance of cyp2d6 polymorphism and or drug interactions to m -cpp toxicokinetics staack et al.
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Following ingestion of trazodone, those with high bmrs are thought to metabolize and excrete the drug more efficiently than those with low bmrs.
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